Photo - dreamstime.com

Video- Miles Teller talks about his reaction from exposure to toxins in a ‘Top Gun’ jet:

https://m.youtube.com/shorts/tTn8-KHUJWc?si=UwgS_gvkJa8XEKi8

The story in the video sounds very funny doesn’t it?, especially Tom Cruise’s response. 
Aircrew working onboard commercial jets are exposed to those chemicals every time they go to work and reactions to the toxins in aircraft cabin air can occur at any time. 

Miles Teller is very fortunate that he chose to be an actor and not an airline pilot. 

TILT -
‘Toxicant-induced loss of tolerance (TILT) is a two-stage disease mechanism first described in the 1990s [1, 2] and characterized by multisystem symptoms and new-onset intolerances that develop in a subset of individuals following major chemical exposure events or repeated low-level exposures.’
https://enveurope.springeropen.com/articles/10.1186/s12302-021-00504-z

Sadly, the pilot in the case study below lost his commercial pilot’s license and flying career which has happened to so many pilots. Others, as well as cabin crew have lost their lives.
Prolonged Disability following Re-Exposure after Complete Recovery from Aerotoxic Syndrome: A Case Report (December 2023)
by Race Creeden Nick Blonien 
Jill K. Schultz Jessika Wheeler 
Erik L. Haltson Zeke J. McKinney 
https://www.mdpi.com/1660-4601/20/24/7156

 Toxic Triggers
Exposure Assessment and Timeline
of the gradual loss of tolerance to chemical substances.
Teaching Paper – Case Study
Bearnairdine Beaumont (January 2021)

‘Abstract
Toxic exposure can have occurred unnoticed for many years in the past—some toxic substances deposit in the organs and bones, and others in fatty tissues. The presence of synthetic chemicals in the body's fat cells slows down the natural detoxification process. Over time, this can lead to the body carrying a toxic load, also known as toxic body burden. Chemicals vary widely in the timescale it takes for their potency to reduce - known as the chemical half-life.
Even at low levels, chronic exposure to toxic substances increases the body's toxic inflammatory reaction. The body will do its best to deal with foreign or 'xenobiotic' invaders. However, at some stage, it will run out of steam and ill-health results.
A person's susceptibility to chemicals depends on many factors. Some cases by the genes they inherit and others by epigenetic effects that can be changed by environmental factors. Whether a person is ill or not and whether a toxic event has happened in the organism can be shown scientifically by finding a metabolite or original substance in human biomonitoring or fatty tissue biopsies. Alternatively, by provocation tests. A validated analysis and exposure assessment that explicitly prove the relationship of the substance type or noxious agent (pollutant) and the provocation effects are required.’
https://www.academia.edu/44967611/Toxic_Triggers_Exposure_Assessment_and_Timeline_of_the_gradual_loss_of_tolerance_to_chemical_substances_Teaching_Paper_Case_Study

SENSITISATION: CLUE TO THE PATHOLOGY PATHWAY OF CHRONIC ORGANOPHOSPHATE POISONING
A REVIEW OF THE EVIDENCE WITH CASE REPORTS
Helen Fullerton PhD . Summary (1999)

‘The marker of chronic OP poisoning is sensitisation : a heightened sensitivity to low exposures of the initiating agent and cross sensitisation or multiple chemical sensitivity (MCS) to a range of unrelated chemicals, including other pesticides, solvents, perfumes and certain foods. Sensitisation can be defined as chemical kindling and is intertransferable with experimental electrical kindling. Kindling is an amplification process where the initial stimulus has no effect. but repetitive stimulation of a brain area, usually in the limbic system induces abnormal discharges and release of glutamate at high frequencies, mediated by N-Methyl-D-Aspartate (NMDA) receptors. Electrical kindling culminates in seizures. Chemical kindling induces dysfunction of sensory reception and emotional responses via the amygdala. impairment of memory and cognition via the hippocampus and interference with odour discrimination via the olfactory lobe. Alterations in motor, endocrine, autonomic and immune function are relayed through the hypothalamus, midbrain and brain stem.
Corticoid releasing hormone (CRH), released in response to stress is essential to the amplification of the excitatory stimulus. Chemical kindlers powerful enough to induce a biochemical stress releasing CRH include GABA antagonists, such as the convulsants bicucullin and picrotoxin, excitatory amino acid (EAA) agonists, opiod peptides, certain chlorinated hydrocarbons, such as dieldrin and lindane, local anaesthetics, cholinergic agonists and acetyl cholinesterase (AChE) inhibitors, notably the organophosphate (OP) and carbamate pesticides.
Kindling is time-dependent ie. time is required between each electrical stimulation or chemical exposure to allow build-up of after-discharges. Hence chemical sensitisation is not due to cumulative toxicity, which would correspond to continuous stimulation and lead in the case of poisons to acute toxicity, or sometimes in the case of toxic irritants to adaptation.
Sensitisation and cross-sensitisation are initiated in the olfactory lobe. OPs are volatile in hot weather or when carried by evaporating solvent. Volatile molecules that by-pass the blood vessels in the nasal epithelim have rapid access through the olfactory lobe via transneural transport to most areas of the brain: the cortex. the limbic structures where repeated exposures to low levels induce chemical kindling, and to all of their connections. In the olfactory lobe itself the first and second order neurons are dominated by excitatory ACh whose role is to control CRH and glutamate release by activating inhibitory GABA. In the presence of OP there is down-regulation of cholinergic receptors and inhibitory control may be insufficient to prevent a CRH-amplified release of glutamate. The result is an intolerant response to certain odours.
Cross sensitisation occurs because short chain volatiles such as formaldehyde, acetone etc. stimulate release of glutamate. Hence those with MCS react to solvents, perfumes or any substance containing short chain volatiles with a hypersensitive release of glutamate and onset of symptoms similar to those induced by the OP itself.’
http://www.oprus2001.co.uk/fullertonops.pdf

The organophosphates used in the operation of aircraft can cause the same effects on human health as organophosphate pesticides. 
They are also confirmed to be endocrine disrupting chemicals and are known to cause mitochondrial dysfunction as well as cytotoxicity and genotoxicity. 
117 of the chemicals detected in aircraft cabin air to date, are endocrine disrupting chemicals. 

There really isn’t anything cool about being exposed to these chemicals or having them in your body. 

Fortunately, Miles seems to have made a complete recovery since his exposure while working on the film. 

For further information on Aerotoxic Syndrome, useful medical information, testimonies from victims, Aerotoxic legal cases and much more - please visit:
https://www.unfiltered.vip/

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