Please see the following letter addressed to the TBD Working Group regarding the request to create a subcommittee to study the mishandling of Lyme disease. Anyone wishing to contact the Lyme Disease Working Group can send an email to: tickbornedisease@hhs.gov Letter to the Working Group: -------- Original Message ---------- From: Carl Tuttle To: tickbornedisease@hhs.gov Cc: Date: March 2, 2018 at 10:03 AM Subject: Is the risk of early neurologic Lyme borreliosis reduced by preferentially treating patients with erythema migrans with doxycycline? ScienceDirect Is the risk of early neurologic Lyme borreliosis reduced by preferentially treating patients with erythema migrans with doxycycline? FrancStrle DašaStupica PetraBogovič PaulVisintainer Gary P.Wormser Available online 2 February 2018 https://www.sciencedirect.com/science/article/pii/S0732889318300427 Excerpt: “Nineteen studies were reviewed to determine if treatment of patients with erythema migrans with other oral antibiotics would increase the risk for developing NLB.” To the TBD Working Group, Dr. Gary Wormser’s latest paper is additional evidence that he has had a fixation on the acute stage of disease after erythema migrans (Bulls-eye rash) excluding the sickest of the patient population as I have identified in an earlier complaint registered with the Office of Research Integrity. 2015 complaint to the attention of Don Wright, M.D., M.P.H. https://www.dropbox.com/s/94jv3w2hb0z9lv7/Email%20sent%20to%20the%20Office%20of%20Research%20Integrity.docx?dl=0 This is the equivalent of refusing to acknowledge that untreated strep throat progresses to rheumatic fever which is entirely different disease causing irreversible heart damage. Patients with a prolonged exposure to a Borrelia infection before diagnosis and treatment are almost always incapacitated. What happens to the Lyme patient who went months, years or decades with an untreated spirochetal infection? Perhaps we should ask the Centers for Disease Control for their findings of the Tuskegee Airman experiment. The Tuskegee Timeline from the CDC website: https://www.cdc.gov/tuskegee/timeline.htm Take a look at the autopsy report of Lyme patient Vicki Logan from the attached letter to Brenda Fitzgerald, Director of the Centers for Disease Control. Logan’s autopsy report identifies a nutmeg liver. How does a patient recover from a nutmeg liver? Letter to Brenda Fitsgerald, Director of the Centers for Disease Control https://www.dropbox.com/s/xaul84dqmqgbre0/Brenda%20Fitzgerald%20MD%20Director%20CDC.docx?dl=0 And what about the published case study from Stony Brook Lyme Clinic reporting Seronegative Chronic Relapsing Neuroborreliosis as this patient experienced; “repeated neurologic relapses despite aggressive antibiotic therapy.” Is this case acknowledged in Wormser’s latest published paper? Seronegative Chronic Relapsing Neuroborreliosis. https://www.ncbi.nlm.nih.gov/pubmed/7796837 _____________________________ How serious is Lyme? Per the 1988 paper below co-authored by Allen Steere, you will see that Lyme isn’t just the “aches and pains of daily living.” This paper was published before the rush to develop a vaccine for Lyme disease. When the vaccine was conceived, suddenly Lyme became a simple nuisance disease. Clinical pathologic correlations of Lyme disease by stage. (I recommend everyone read this TWICE!) http://www.actionlyme.org/clinical-pathologic-correlations-of-lyme-disease-by-stage-Steere-Duray.pdf Abstract: Lyme disease is capable of producing a wide variety of clinical pathologic conditions and lesions having in common histologic features of collagen-vascular disease. The plasma cell is an omnipotent inflammatory responder in most tissues involved by Lyme disease, ranging from relatively acute to lesions that have gone on for years. Vascular thickening also seems to be prominent, and in the dermis is accompanied by scleroderma-like collagen expansion. The disease in some ways resembles the responses seen in lupus erythematosus such as mild cerebritis with lymphocytes and plasma cells in the leptomeninges. Lymphoplasmacytic panniculitis of Lyme disease resembles lupus profundus, both in the infiltrate and the plasma cell-blood vessel relationship. The onion skin thickened vessels of the synovia resemble the vessels of lupus spleens, while the scleradermoid thickening of the dermis and various skin lesions of stage III Lyme disease suggest a collagen-vascular disorder. Finally, the perivascular lymphoid infiltrate in clinical myositis does not differ from that seen in polymyositis or dermatomyositis. All of these histologic derangements suggest immunologic damage in response to persistence of the spirochete, however few in number. ________________ I believe Steere recognized an EBV-like induced lymphoma; as you all know I am fighting right now as a result of undiagnosed Lyme of twelve years. Wormser’s decades of junk science has deliberately and purposely suppressed evidence of persistent infection/seronegative disease while he compares Lyme disease with its life-altering/life-threatening consequences to the aches and pains of daily living. A thirty year racketeering scheme as outlined in the RICO lawsuit. Scientific misconduct to support one’s bias is fraud which has had an impact on public health and clinical treatment. Court document: https://www.courthousenews.com/wp-content/uploads/2017/11/LymeDisease.pdf Where is the Lyme disease treatment guideline for the patient who has progressed to nutmeg liver? Might I remind everyone reading this email that the Centers for Disease Control has aligned itself with Dr. Gary Wormser so do we want this duo involved in the future direction of Lyme disease diagnostics and treatment? I have presented evidence that the CDC has avoided direct detection methods for the diagnosis of Lyme disease. An accurate test for the detection of Lyme in all stages of disease will foil the existing racketeering scheme exposing just how wrong the CDC has summed up the disease for nearly four decades. Right now, serology cannot gauge treatment failure or success which makes antibody tests the ideal tool for concealing persistent infection. The removal of outer surface protein A and B (Bands 31 and 34 of the Western blot) for the purpose of vaccine development is a crime. As I mentioned in the previous email to the Working Group, a subcommittee should be assigned to study the deliberate mishandling of Lyme disease and recommend the removal of the Wormser/CDC team in any future involvement with diagnostics and treatment of Lyme disease. Turning a blind eye to the fraudulent handling of Lyme disease is support of the offense. Respectfully submitted, Carl Tuttle Hudson, NH “Justice will not be served until those who are unaffected are as outraged as those who are.” ― Benjamin Franklin Cc: Daniel R. Dutko, HANSZEN LAPORTE